Hiv Research Papers

That could include a correction, a revised and annotated journal article, or a full retraction.

"It's more a question of imprecise use of language or an assumption that people would understand what it meant, even though it was not explicitly stated in the paper," Berg said.

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In contrast, the monkeys in the control arm all progressed and died of the virus.

Those findings prompted Anthony Fauci, MD, director of the National Institute of Allergy and Infectious Diseases (NIAID), to call the findings "striking" at the HIV Research for Prevention conference in October 2016.To learn more and make choices about data use, visit our Advertising Policy and Privacy Policy.By clicking “Accept and Continue” below, (1) you consent to these activities unless and until you withdraw your consent using our rights request form, and (2) you consent to allow your data to be transferred, processed, and stored in the United States."If you have to place animals on [antiretroviral treatment] before 2 weeks because the virus is too virulent (ie, a study I am currently involved in), to ensure animals have an immune response worth salvaging therapeutically, the value of such a model is, in my mind, even more questionable," he told Yet "we did not have any animals that totally suppressed SIV in the absence of antiretroviral drug," he said.In a presentation of that study at the 2018 International AIDS Conference, the researchers make clear that they used the gene from all the monkeys to see whether the stop codon was still present in the monkeys before they began antiretroviral therapy and then the anti-alpha4beta7 treatment. "I'm the first to admit that something funny is going on.If the virus had repaired itself and the stop codon was gone, Ansari said, perhaps there's some mechanism, some dynamic between the monkeys' own immune systems and the anti-alpha4beta7 treatment that could still offer hope as an avenue of investigation. But I'm trying to point out the fact that [NIAID] got the virus from Villinger and got different results." "I'm reluctant to say any field is dead; alpha4beta7 is an important pathway in HIV infection," he said."But whether an antibody to alpha4beta7 can lead to consistent, long-term suppression of the virus — that is clearly now questionable." Comments on Medscape are moderated and should be professional in tone and on topic.Berg told that he received tips from a variety of directions, starting at the beginning of the year, that the virus used in the study that started all of this wasn't exactly what the published paper said it was.One person who knew exactly what virus the 2016 study used, as well as the follow-up nonhuman primate study that wasn't successful, was Francois Villinger, DVM, of the University of Louisiana at Lafayette.He's the one who provided the virus for both animal studies.The conflict, then, isn't over whether the mutated version of SIVmac239 was used and not specifically stated in the paper.

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